@article{3087399,
    title = "New evidence of gene inactivation by aberrant DNA-Methylation in T-cell leukemia, with treatment implications",
    author = "Kyrtsonis, M.-C. and Maltezas, D.",
    journal = "Leukemia Research",
    year = "2016",
    volume = "50",
    pages = "29-30",
    publisher = "Elsevier Ireland Ltd",
    issn = "0145-2126",
    doi = "10.1016/j.leukres.2016.09.010",
    keywords = "azacitidine;  decitabine;  DNA;  DNA methyltransferase 1;  DNA methyltransferase 3A;  histone deacetylase;  romidepsin;  vorinostat;  DNA, acute myeloblastic leukemia;  cancer chemotherapy;  cancer combination chemotherapy;  cancer growth;  cancer prognosis;  cancer recurrence;  cancer risk;  cancer survival;  clinical outcome;  clinical trial (topic);  cutaneous T cell lymphoma;  demethylation;  DNA methylation;  drug mechanism;  drug response;  Editorial;  enzyme inhibition;  epigenetics;  evidence based practice;  gene expression regulation;  gene identification;  gene inactivation;  gene silencing;  human;  jurkat cell line;  leukemogenesis;  malignant transformation;  multiple myeloma;  mycosis fungoides;  myelodysplastic syndrome;  peripheral T cell lymphoma;  priority journal;  pyrosequencing;  Sezary syndrome;  T cell leukemia;  T-cell leukemia cell line;  tumor suppressor gene;  DNA methylation;  gene silencing;  genetics;  methylation;  promoter region;  T cell leukemia, DNA;  DNA Methylation;  Gene Silencing;  Humans;  Leukemia, T-Cell;  Methylation;  Promoter Regions, Genetic"
}